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The practice of equine dentistry is an integral branch of equine veterinary medicine. This discipline encompasses all aspects of diagnosis, treatment, and prophylaxis of any and all equine dental conditions and diseases that affect the oral cavity, mandible and maxilla teeth and associated structures. As such, it falls within the purview of veterinary medicine. Any surgical procedure of the head or oral cavity; the administration or prescription of sedatives, tranquilizers, analgesics or anesthetics; procedures which are invasive of the tissues to the oral cavity including, but not limited to, removal of sharp enamel projections, treatment of malocclusions of premolars, molars, and incisors, reshaping of canine teeth, the extraction of first premolars and deciduous premolars and incisors; treatment, extraction or repair of damaged or diseased teeth; periodontal treatment; and dental radiography are veterinary medical procedures and should be performed by a licensed veterinarian. In states where the Veterinary Practice Act allows, the AAEP supports the use of licensed veterinary technicians under the employ and supervision of licensed veterinarians for specific and appropriate veterinary dental procedures as enumerated in that state's practice act.
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Concentrations Ki 1.05 M ; , while tolterodine immediate release as well as the 5-hydroxymethyl metabolite are devoid of any significant inhibitory potential regarding the other isoenzymes. CYP3A4 Inhibitors: The effect of a 200-mg daily dose of ketoconazole on the pharmacokinetics of tolterodine immediate release was studied in 8 healthy volunteers, all of whom were poor metabolizers see Pharmacokinetics, Variability in Metabolism for discussion of poor metabolizers ; . In the presence of ketoconazole, the mean Cmax and AUC of tolterodine increased by 2 and 2.5 fold, respectively. Based on these findings, other potent CYP3A4 inhibitors such as other azole antifungals eg, itraconazole, miconazole ; or macrolide antibiotics eg, erythromycin, clarithromycin ; or cyclosporine or vinblastine may also lead to increases of tolterodine plasma concentrations see PRECAUTIONS and DOSAGE AND ADMINISTRATION ; . Warfarin: In healthy volunteers, coadministration of tolterodine immediate release 4 mg 2 mg bid ; for 7 days and a single dose of warfarin 25 mg on day 4 had no effect on prothrombin time, Factor VII suppression, or on the pharmacokinetics of warfarin. Oral Contraceptives: Tolterodine immediate release 4 mg 2 mg bid ; had no effect on the pharmacokinetics of an oral contraceptive ethinyl estradiol 30 g levonorgestrel 150 g ; as evidenced by the monitoring of ethinyl estradiol and levonorgestrel over a 2-month period in healthy female volunteers. Diuretics: Coadministration of tolterodine immediate release up to 8 mg 4 mg bid ; for up to 12 weeks with diuretic agents, such as indapamide, hydrochlorothiazide, triamterene, bendroflumethiazide, chlorothiazide, methylchlorothiazide, or furosemide, did not cause any adverse electrocardiographic ECG ; effects. CLINICAL STUDIES DETROL LA Capsules 2 mg were evaluated in 29 patients in a Phase 2 dose-effect study. DETROL LA 4 mg was evaluated for the treatment of overactive bladder with symptoms of urge urinary incontinence and frequency in a randomized, placebocontrolled, multicenter, double-blind, Phase 3, 12-week study. A total of 507 patients received DETROL LA 4 mg once daily in the morning and 508 received placebo. The majority of patients were Caucasian 95% ; and female 81% ; , with a mean age of 61 years range, 20 to 93 years ; . In the study, 642 patients 42% ; were 65 to 93 years of age. The study included patients known to be responsive to tolterodine immediate release and other anticholinergic medications, however, 47% of patients never received prior pharmacotherapy for overactive bladder. At study entry, 97% of patients had at least 5 urge incontinence episodes per week and 91% of patients had 8 or more micturitions per day. The primary efficacy endpoint was change in mean number of incontinence episodes per week at week 12 from baseline. Secondary efficacy endpoints included change in mean number of micturitions per day and mean volume voided per micturition at week 12 from baseline. INDICATIONS AND USAGE DETROL LA Capsules are once daily extended release capsules indicated for the treatment of overactive bladder with symptoms of urge urinary incontinence, urgency, and frequency. CONTRAINDICATIONS DETROL LA Capsules are contraindicated in patients with urinary retention, gastric retention, or uncontrolled narrow-angle glaucoma. DETROL LA is also contraindicated in patients who have demonstrated hypersensitivity to the drug or its ingredients. PRECAUTIONS General Risk of Urinary Retention and Gastric Retention: DETROL LA Capsules should be administered with caution to patients with clinically significant bladder outflow obstruction because of the risk of urinary retention and to patients with gastrointestinal obstructive disorders, such as pyloric stenosis, because of the risk of gastric retention see CONTRAINDICATIONS.
Shimoyama N, et al. Nebulized furosemide as a novel treatment for dyspnea in terminal cancer patients. J Pain Symptom Manage. 2002; 23 1 ; : 73 Patient Population Profile Patient 1: 49-yearold male patient with bladder cancer, lung metastases, and severe dyspnea. Received morphine infusion 100 mg daily with 4 mg IV rescue doses ; , but dyspnea was unrelieved. Dyspnea was rated 8.5 10 on VAS. Also received no relief with O2 therapy via nasal cannula and nebulized orciprenaline [metaproterenol] 4 mg 4 times daily ; . Drug Route ; Dose Duration ; Furosem9de NEB ; : 20 mg 4 times daily diluted in 2 mL normal saline for total volume of 4 mL. Each nebulized episode lasted 10 to 15 minutes. Results Safety Author Conclusions.

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If your problem persists even after changing nail-care products, it may be best to see a physician to be sure your problem is not an underlying health issue for you. T.D.J. Smilde 1 , M. Zuurman 2 , H.L. Hillege 1 , F.W. Asselbergs 1 , A.A. Voors 1 , J.A. Kors 3 , D.J. Van Veldhuisen 1 , G. Navis 2 on behalf of PREVEND investigators. 1 University Medical Center Groningen, Cardiology, Groningen, Netherlands; 2 University Medical Center Groningen, Nephrology, Groningen, Netherlands; 3 Erasmus University Medical Center, Cardiology, Rotterdam, Netherlands Background: Worsening of renal function is associated with an increased preva and gemfibrozil. Generic Name 7. DIURETICS 7.1 Diuretics acetazolamide amiloride amiloride hydrochlorothiazide bumetanide chlorothiazide chlorthalidone furosemide hydrochlorothiazide indapamide methazolamide methyclothiazide metolazone spironolactone spironolactone hydrochlorothiazide torsemide triamterene & hydrochlorothiazide triamterene & hydrochlorothiazide triamterene & hydrochlorothiazide 75 50 8. LIPID LOWERING AGENTS 8.1 Lipid Lowering Agents cholestyramine colestipol fenofibrate fenofibrate gemfibrozil 8.2 HMG CoA Reductase Inhibitors ST ezetimibe-simvastatin QL lovastatin QL lovastatin suspended release simvastatin 9. NITRATES 9.1 Nitrates isosorbide dinitrate isosorbide dinitrate suspended release isosorbide mononitrate isosorbide mononitrate suspended rel. nitroglycerin nitroglycerin lingual spray. 3.1.4.2 Plot Location, Fragmentation and Size The predominance of mixed-cropping culture and the increased fragmentation of land in the study area make the definition of a plot very difficult. Here we followed the definition that a plot, which is equivalent to saying a parcel, is a piece of land covered by a single cropping system. The cropping system could be both mono-cropping as well as mixed cropping. Most plots operated by the sample households are located around the homesteads and are referred to as compound farms. The compound farms account for about 60 percent of the plots followed by bushes, which account for about 20 percent of the plots. Even under the compound and bush plots farm households operate fragmented plots. 42 percent of the sample households operate more than 4 plots of land and 25 percent of households have exactly four plots Table 3.4 and glucophage, for instance, furosemide drug.

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I swore i would never have that drug if i needed to be induced. Note that medications that use multiple words are linked by underscores to preserve the entire name in Text Miner. Text Miner is then used on the re-defined dataset to find the relationships on those text strings. The clusters are given in Table 4. below. By using a combination of medications that are linked, the optimal number of clusters as identified in Text Miner is equal to 21. Once the clusters are defined, they can be used with other statistical analyses. Association is preserved in the linkage defined by the SAS code. The Association Node can no longer be used with this dataset as each customer now has only one record. Attempts to run the Association Node will result in a finding of zero rules. Table 4. Clusters of Medications Cluster Cluster Description Number 1 glucophage, furosemide, zestril, synthroid, softclix 2 allegra, claritin, flonase, nasonex, hydrochlorothiazide 3 lipitor, vioxx, allegra, claritin 4 augmentin 5 vicodin, apap hydrocodone bitartrate, celexa, naproxen, vioxx 6 zoloft, triple antibiotic, paxil, zyrtec, naproxen and glucotrol. 363AMBIEN1OMG TABLET TAB ROCODON E APAP5 5OO 359I-IYD 20 FUROSEMIDE MGTABLET CP 37.5125 334 TRIAMTERENE HCTZ 20 333LIPITOR MGTABLET 40 EC 304 PROTONIX MG TABLET 292 LISINOPRIL MG TABLET 40 287 ACTONEL MGTABLET 35.

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HOW SUPPLIED LASIX furosemide ; Tablets 20 mg are supplied as white, oval, monogrammed tablets in Bottles of 100 NDC 0039-0067-10 ; , 500 NDC 0039-0067-50 ; , and 1000 NDC 0039-0067-70 ; . The 20 mg tablets are imprinted with "Lasix" on one side and "HOECHST" on the other, or with "Lasix" on one side. LASIX Tablets 40 mg are supplied as white, round, monogrammed, scored tablets in Bottles of 100 NDC 0039-0060-13 ; , 500 NDC 0039-0060-50 ; , 1000 NDC 0039-0060-70 ; , and Unit Dose Packs of 100 NDC 0039-0060-11 ; . The 40 mg tablets are imprinted with "Lasix 40" on one side and the Hoechst logo on the other, or with "Lasix 40" on one side. LASIX Tablets 80 mg are supplied as white, round, monogrammed, facetted edge tablets in Bottles of 50 NDC 0039-0066-05 ; and 500 NDC 0039-0066-50 ; . The 80 mg tablets are imprinted with "Lasix 80" on one side and the Hoechst logo on the other, or with "Lasix 80" on one side. Note: Dispense in well-closed, light-resistant containers. Exposure to light might cause a slight discoloration. Discolored tablets should not be dispensed. Tested by USP Dissolution Test 2 Store at 25 C excursions permitted to 15 - 30 [See USP Controlled Room Temperature.] Revised June 2006 Manufactured for: sanofi-aventis U.S. LLC Bridgewater, NJ 08807 2006 sanofi-aventis U.S. LLC and glyburide.

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Addition of these agents to California's Medi-Cal drug formulary was correlated with an overall improvement in the outcome of drug therapy and a corresponding decrease in hospital and nursing home costs. However, since drug expenditures rose, total expenditures were unchanged. Although overall costs were not reduced, the addition of these second-generation agents resulted in improved glycemic control without increasing costs.26.

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No-one knows who will be affected by the disorder but if this is suspected then it is important to get the relevant medical advice immediately because there can be serious consequences if bipolar disorder goes untreated, for example, furosemide use. 1162 FUNDUS PHOTOGRAPHY. SUCCESS FOR SCREENING AND LIMITATIONS FOR CHARACTERISATION OF THE INITIAL STAGES OF D.R. SUMMANEN PA 1, 2 ; , VON WENDT GC 2, 3 ; 1 ; Department of Ophthalmology, Helsinki University Central Hospital, Helsinki, Finland 2 ; Diabetic Retinopathy Photographic Screening Unit, Helsinki City Health Dept, 3 ; St. Erik's Eye Hospital, Stockholm, Sweden Diabetic retinopathy DR ; fulfills all the criteria for a state to be screened; 1 ; target population is well defined, 2 ; economically feasable and sensitive screening methods are available, 3 ; the natural course of the disease is well known, and 4 ; an effective treatment exists. Fundus photo- graphy FP ; is the method of choise for screening and follow up of DR because it is noninvasive, technically easy, gives documentation and is well accepted by patients. Color transparencies, color, and black and white paper-prints and digitalized images using varying number and width of the fields n 1-7, 30-60 degree ; are used, the gold standard being the stereoscopic 7-field, 30 degree FP used in many large-scale randomized clinical trials e.g. DRS, ETDRS ; . Initial changes in DR are haemorrhages and or microaneuryms H Ma ; , venous loops, microinfarctions, lipid exudates, intraretinal microvascular abnormalities IRMA ; and venous beading VB ; , all of which occur in several levels of severity and combinations, and define the severity level of DR. Green filter improves the detection of the most crucial early changes, H Ma, IRMA, and VB, and even the smallest new vessels on the disc and or retina, because the hemoglobin-containing structures are well seen. It almost equals fluorescein angiography in revealing structural, but not functional, changes of the vasculature of the ocular fundus and hydrocodone.
Furosemide oral solution 4mg ml Expiry : 25.09.07. Additionally, some medications will break down into harmful byproducts with the passage of time and hyzaar. 1 2 3 Holz, Jr., G. G. 1977 ; Bull. World Health Org. 55, 237248 Ancelin, M. L. and Vial, H. J. 1989 ; Biochim. Biophys. Acta 1001, 8289 Martin, K. 1977 ; in Membrane Transport in Red Cells Ellory, J. C. and Lew, V. L., eds. ; , pp. 101113, Academic Press, London Ancelin, M. L., Parant, M., Thuet, M. J., Philippot, J. R. and Vial, H. J. 1991 ; Biochem. J. 273, 701709 Kirk, K., Wong, H. Y., Elford, B. C., Newbold, C. I. and Ellory, J. C. 1991 ; Biochem. J. 278, 521525 Kirk, K., Horner, H. A., Elford, B. C., Ellory, J. C. and Newbold, C. I. 1994 ; J. Biol. Chem. 269, 33393347 Kirk, K. and Horner, H. A. 1995 ; J. Biol. Chem. 270, 2427024275 Cabantchik, Z. I. 1990 ; Blood Cells 16, 421432 Ginsburg, H. 1994 ; Biochem. Pharmacol. 48, 18471856 Kirk, K. and Horner, H. A. 1995 ; Biochem. J. 311, 761768 Elford, B. C., Cowan, G. M. and Ferguson, D. J. 1995 ; Biochem. J. 308, 361374 Ancelin, M. L. and Vial, H. J. 1992 ; Biochem. J. 283, 619621 Kirk, K., Horner, H. A. and Kirk, J. 1996 ; Mol. Biochem. Parasitol. 82, 195205 Ellory, J. C., Kirk, K., Culliford, S. J., Nash, G. B. and Stuart, J. 1992 ; FEBS Lett. 296, 219221 Das, I., de Belleroche, J., Moore, C. J. and Rose, F. C. 1986 ; Anal. Biochem. 152, 178182 Singh, S., Puri, S. K., Singh, S. K., Srivastava, R., Gupta, R. C. and Pandey, V. C. 1997 ; J. Biol. Chem. 272, 1350613511 Kirk, K., Poli de Figueiredo, C. E., Elford, B. C. and Ellory, J. C. 1992 ; Biochem. J. 283, 617619 Crandall, I. and Sherman, I. W. 1991 ; Parasitology 3, 335340 Maguire, P. A., Prudhomme, J. and Sherman, I. W. 1991 ; Parasitology 2, 179186 Homewood, C. A. and Neame, K. D. 1974 ; Nature London ; 252, 718719 Gati, W. P., Lin, A. N., Wang, T. I., Young, J. D. and Paterson, A. R. P. 1990 ; Biochem. J. 272, 277280 Upston, J. M. and Gero, A. M. 1995 ; Biochim. Biophys. Acta 1236, 249258 Saliba, K. J., Horner, H. A. and Kirk, K. 1998 ; J. Biol. Chem. 273, 1019010195.
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Furosemide-induced renal medullary hypoperfusion in the rat: role of tissue tonicity, prostaglandins and angiotensin II Leszek Dobrowolski and Janusz Sadowski J. Physiol. 2005; 567; 613-620; originally published online Jun 16, 2005; DOI: 10.1113 jphysiol.2005.090027 This information is current as of July 24, 2007 and imitrex and furosemide.

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Figure 2. Graph showing plasma renin activity PRA ; values obtained in the 3 dietary groups of stroke-prone spontaneously hypertensive rats at 4-week intervals. Open circles indicate regular diet n 7, week 0; n 6, week 4; n 6, week 8; n 13, week 12 open squares, low-NaCl low potassium n 8, week 4; n 13, week 8; n 11, week 12 and open triangles, high-NaCl lowpotassium diet n 9, week 4; n 10, week 8; n 14, week 12 ; . Note that plasma renin "escapes" from suppression by salt in the group destined for vascular injury and stroke. From Volpe M, Camargo MJF, Mueller FB, Campbell WG Jr, Sealey JE, Pecker MS, Sosa RE, Laragh JH. Hypertension. 1990; 15: 318 Laragh JH, Heinemann HO, Demartini FE. The effect of chlorothiazide on electrolyte transport in man: its use in the treatment of edema of congestive heart failure, nephrosis and cirrhosis. JAMA. 1958; 166: 145152. Laragh JH. Spironolactone for treatment of hypertension or congestive heart failure: a review. Excerpta Medica. Princeton, NJ: Princeton University Press; 1987. 3. Pitt B, Zannad F, Remme WJ, Cody R, Castaigue A, Perez A, Palensky J, Wittes J. The effect of spironolactone on morbidity and mortality in patients with severe heart failure. N Engl J Med. 1999; 341: 709 Dyckner T, Wester P-O, Widman L. Effects of spironolactone on serum and muscle electrolytes in patients on long-term diuretic therapy for congestive heart failure and or arterial hypertension. Eur J Clin Pharmacol. 1986; 30: 535540. Dyckner T, Wester P-O. Plasma and skeletal muscle electrolytes in patients on long-term diuretic therapy for arterial hypertension and or congestive heart failure. Acta Med Scand. 1987; 222: 231236. Dyckner T. Relation of cardiovascular disease to potassium and magnesium deficiencies. J Cardiol. 1990; 65: 44K Cody RJ, Covit AB, Schaer GL, Laragh JH, Sealey JE, Feldschuh J. Sodium and water balance in chronic congestive heart failure. J Clin Invest. 1986; 77: 14411452. Cannon PJ, Ames RP, Laragh JH. Relation between potassium balance and aldosterone secretion in normal subjects and in patients with hypertension or renal tubular disease. J Clin Invest. 1966; 45: 865 Laragh JH, Cannon PJ, Stason WB, Heinemann HO. Physiologic, and clinical observations on furosemide and ethacrynic acid. Ann NY Acad Sci. 1966; 139: 453 Davis JO. The physiology of congestive heart failure. In: Hamilton WF, ed. Handbook of Physiology II: Circulation, Vol 3. Washington, DC: American Physiology Society; 1965; 20712122. 11. Sealey JE, Laragh JH. A proposed cybernetic system for sodium and potassium homeostasis: coordination of aldosterone and intrarenal physical factors. Kidney Int. 1974; 6: 281290. Weber MA, Drayer JIM, Rev A, Laragh JH. Disparate patterns of aldosterone response during diuretic treatment of hypertension. Ann Intern Med. 1977; 87: 558 Franse LV, Pahor M, DiBari M, Somes GW, Cushman WC, Applegate WB. Hypokalemia associated with diuretic use and cardiovascular events in the Systolic Hypertension in the Elderly program. Hypertension. 2000; 35: 10251030. Multiple Risk Factor Intervention Trial Research Group. Multiple Risk Factor Intervention Trial: risk factor changes and mortality results. JAMA. 1982; 248: 14651477. Multiple Risk Factor Intervention Trial Research Group. Baseline rest electrocardiographic abnormalities, antihypertensive treatment, and mortality in the Multiple Risk Factor Intervention Trial. J Cardiol. 1985; 55: 115. Holland OB, Kuhnert L, Pollard J, Padia M, Anderson RJ, Blomqvis G. Ventricular ectopic activity with diuretic therapy. J Hypertens. 1988; 1: 380 Several biochemical or cellular mechanisms have been implicated to explain how K depletion promotes cardiac and vascular injury, which is correctable by K repletion. Thus, increases in extracellular K levels within the physiological range in 1-mmol L increments from 3 to 7 mmol L causes significant decreases in free radical formation from vascular endothelial cells, smooth muscle proliferation, and induced thrombus formation.24 With verification, these pathways could prove to be relevant clinical targets for pharmacological control. In summary, thiazide-induced K and or Mg2 depletion in hypertension and in CHF is probably not benign. It impairs cardiac function, and it creates or enhances the risk of morbid cardiac and vascular events. In hypertensive patients, these effects of K depletion more than cancel the cardiovascular protective value afforded by the concurrent sizable thiazide. Minoxidil this former prescription medication is now available over the counter for use by both men and women, marketed under the brand name rogaine and isosorbide.
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Post time of the race in which the horse is entered except that furosemide "Lasix" ; may be administered under the supervision of the Official Veterinarian as prescribed in these Rules. e ; When a foreign substance of accepted therapeutic value is administered or prescribed by a veterinarian for a horse that is entered to race, the veterinarian must make a report of that substance and submit the report to the Commission Veterinarian by 9: 00 a.m. of the race day and must report immediately following administration of Lasix. f ; The administration of 250 milligrams of furosemide "Lasix" ; shall be permitted four or more hours prior to post time for the prophylactic treatment of a known bleeder. Stabling until race reporting time in a holding facility approved by the Commission Veterinarian is required. g ; Bute is the only approved non-steroidal anti-inflammatory drug NSAID ; that may be present in a horse's body while it is participating in a race. The presence of more than one NSAID at any test level is forbidden. A horse running on an approved NSAID will be required to run on an approved NSAID in all subsequent races at the meet, unless special permission for the horse to be removed from the "bute" list is granted by the Commission Veterinarian and the Stewards. h ; The test level of phenylbutazone and its metabolites under this rule shall not be in excess of five 5 ; micrograms per milliliter MCG ML ; in the blood. i ; Each and every horse entered to race may be subjected to a veterinary examination for racing soundness and health on race day, not later than two hours prior to official post time for the first race. i ; Such an examination shall be referred to as the "Racing Soundness Exam". ii ; All such examinations shall be conducted in or near the stall to which the animal is assigned and shall be conducted by a veterinarian employed by the Commission or approved by it. iii ; The veterinarian shall keep a continuing health and racing soundness record of each horse examined. j ; Every horse that suffers a breakdown on the race track, in training or in competition, and is destroyed, and every other horse which expires while stabled on the race track shall undergo a postmortem examination at a time and place acceptable to the Commission Veterinarian to determine the injury or sickness which resulted in euthanasia or natural death. i ; The post-mortem examination required under this rule will be conducted by a veterinarian employed by the owner or his trainer in the presence of and in consultation with the Commission Veterinarian. ii ; Test samples must be obtained from the carcass upon which the post-mortem examination is conducted and shall be sent to a laboratory approved by the Commission for testing for foreign substances and natural substances at abnormal levels. When practical samples should be procured prior to euthanasia. iii ; The owner of the deceased horse shall make payment of any charges due the.
Clearly specify "public use" requirements addressed in Section 100310 a ; 3 ; in order to minimize ambiguity and uncertainty. Remove pricing and access issues as grounds for modification or termination of licenses as provided in Section 100306 f ; . Remove or consider more effective and efficient alternatives to Section 100308 b ; , which establishes a direct revenue-sharing policy for CIRM-funded commercialized technologies. Remove or significantly narrow Section 100307, which broadly requires that CIRMfunded inventions must be made available for research purposes at no cost. Clarify, narrow or remove "failure to keep the licensed invention available to the public for research purposes" as grounds for termination of licenses included in Section 100306 f ; . We look forward to working with the ICOC is it finalizes this policy, and we would be happy to further discuss these comments in additional detail. Thank you for your attention to this important matter. AMC OPS 1.755 Emergency Medical Kit See JAR-OPS 1.755 The following should be included in the emergency medical kit carried in the aeroplane: Sphygmomanometer non mercury Stethoscope Syringes and needles Oropharyngeal airways 2 sizes ; Tourniquet Coronary vasodilator e.g. nitro-glycerine Anti-spasmodic e.g. hyoscine Epinephrine 1: 000 Adrenocortical steroid e.g. hydrocortisone Major analgesic e.g. nalbuphine Diuretic e.g. furosemide Antihistamine e.g. diphenhydramine hydrochloride Sedative anticonvulsant e.g. diazepam Medication for Hypoglycaemia hypertonic glucose and or glucagon Antiemetic e.g. metoclopramide Atropine Digoxin Disposable Gloves Bronchial Dilator injectable and inhaled form Needle Disposal Box Catheter A list of contents in at least 2 languages English and one other ; . This should include information on the effects and side effects of drugs carried.
Blood uric acid levels can increase during furosemide treatment, but precipitation of acute gout is rare and gemfibrozil.
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